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Green Tea Catechins Reduce Body Fat and LDL-Oxidation In Clinical Studies The Research Status of Hydroxycitric Acid Hca The Research Status of Chromium.
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Body Burn quantity. SKU: AD04BB Category: Supplements. Description Additional information Active Ingredients Considerations Supporting Research Reviews 6 Description I ntroducing Adëeva Body Burn, the perfect supplement for individuals struggling with weight issues, blood sugar problems, and athletes seeking enhanced workout effects.
Key Ingredients: Chromium: Improves insulin response, reducing carbohydrate conversion to fat and an elevated metabolism, leading to increased fat burning during rest and exercise, by enhancing muscle uptake of amino acids, chromium aids in the development of lean muscle mass, making you stronger and more explosive during athletic activities.
Hydroxycitric Acid from Garcinia Cambogia : This active compound blocks key liver enzymes responsible for converting carbohydrates into fat, resulting in fewer carbohydrate calories stored as fat.
Moreover, hydroxycitric acid acts as a natural appetite suppressant, promoting a feeling of fullness and reducing the likelihood of overeating. It also supports quicker recovery after endurance training, enhancing daily athletic performance.
Additionally, catechins help release fat into the bloodstream, making it easier for muscles to utilize fat for energy during physical activity and exercise. Clinical studies have shown that subjects who did not change their diet or activity patterns experienced over 5 pounds of weight loss over 12 weeks when supplementing with catechins from green tea extract.
Clinical Studies: — Nagao T. Additional information Weight 0. If a number is listed next to the item below, then adhere to the precautionary note explained in the legend at the bottom of the page.
Conditions Pregnancy Breastfeeding or Lactation Kidney failure Renal failure 6 Received an organ transplant of any kind 6 Diabetic taking insulin 6 If the kidney has been removed 6 Kidney clearance problem e. Insulin 6 Hypoglycemic Drugs 5 Receiving chemotherapy treatment 6 Receiving radiation treatment 6 Legend 5 Monitor patient tolerance with supplementation 6 Requires approval from specialist.
Each set was interspersed with 2 min of rest. Fatigue index was defined as the high torque the average of the three consecutive highest repetitions at the beginning of the set minus the low torque, divided by the high torque.
For each of these variables, the average of three sets was used to represent a single visit. Muscle soreness was assessed using a 7-point Likert scale questionnaire for a variety of muscle groups including the gastrocnemius, hamstrings, quadriceps, gluteus maximus, lower back, abdominals, and the whole body Participants were asked to rate their perceived level of muscle soreness at rest as 1 no pain, 2 dull ache, 3 slight pain, 4 moderate pain, 5 painful, 6 very painful, or 7 severe pain.
A comprehensive metabolic panel was conducted to evaluate the safety of and tolerance to the dietary supplement. Blood was also evaluated to examine potential mechanisms of action of the treatment through the evaluation of various markers of inflammation IL-6, IL, TNF-α , oxidative stress 8-isoprostane, FRAP , muscle stress LDH, CK , and muscle catabolism cortisol and adrenocorticotropic hormone.
Accordingly, IL-6, IL, and TNF-α were assayed from frozen serum samples using an immunoassay with fluorescent detection Luminex Labmap , Lincoln, St. Charles, MO, USA. LDH and CK were analyzed from frozen serum using an immunoassay with chemiluminescent detection Beckman Coulter DXC, Brea, CA, USA.
Cortisol and adrenocorticotropic hormone were analyzed using immunoassay chemiluminescent detection on an Immulite Siemens, Tarrytown, NY, USA. FRAP, which measures the antioxidant capacity of the blood, was measured by a colorimetric assay Beckman Coulter DXC ; and 8-isoprostate was measured by enzyme immunoassay Tarrytown, NY, USA.
The two active study groups consumed different doses of PB Kemin Foods, L. The nutritional analysis for the polyphenol blend extract is shown in Table 2. The study product was encapsulated in gelatin capsules and packaged in light-resistant plastic bottles Five Star Compounding Pharmacy, Des Moines, IA, USA.
The product lots were tested for toxins including heavy metals, pesticides, and excipients. Stability of the capsules was confirmed throughout the study period by measurement of the active components. To consume the required dose, participants ingested 2 capsules twice per day with a meal.
Participants returned at the end of each month to obtain new supplements, and capsules were counted against a known quantity of administered treatment capsules in order to assess compliance. Participants were required to maintain the same exercise and dietary habits during the intervention as reported during screening and were contacted on a weekly basis by phone or email to ask about any adverse events, and encourage compliance and maintenance of exercise and dietary habits.
Analysis of the data for all variables during baseline testing pre-supplementation revealed no differences between groups. Therefore, each outcome variable value used to assess patient recovery was transformed to reflect the change from pre-exercise to post-exercise during the post-supplementation period following 12 weeks of supplementation.
Each transformed variable was analyzed using a mixed model repeated measures MMRM analysis of variance containing terms for treatment, visit, and the treatment×visit interaction.
Least squares means were generated from the model for both main effects and the interaction. Finally, significance levels were determined for main effects, interactions, and pairwise comparisons of each treatment group against the placebo.
One additional analysis was performed to compare serum antioxidant status as measured by FRAP. Using a MMRM model to assess change values between corresponding time points preceding and following the week supplementation period, a contrast from the MMRM model was formed to compare the pooled treatment groups to the placebo between the first available FRAP measurement prior to week supplementation and the first available FRAP measurement post—week supplementation.
The statistical analysis was completed by Summit Analytical, LLC Denver, CO, USA. One hundred twenty-eight participants were screened, of which 63 were eligible for baseline measurements based on inclusion and exclusion criteria, 48 completed baseline testing, and 39 were randomized.
Thirteen individuals from each group completed the intervention period; however, the study physician withdrew one individual from the PB-H group during the follow-up testing period due to clinically elevated CK levels post-exercise.
The final evaluable number for analyses was 12 per group for the PB-L and PB-H groups and 13 in the placebo group. A CONSORT schematic outlining the overall study is provided Fig. CONSORT diagram. Participant screening through study completion is shown for all study participants.
The overall analysis of variance model for peak torque was significant for treatment. Similarly, participants supplemented with PB-H showed no significant decreases in the peak torque values at any time point 24, 48, or 96 h post-exercise compared to pre-exercise. No differences were identified in low torque, total work, average power, or fatigue index following supplementation with PB versus the placebo group.
Biodex peak torque following chronic supplementation. Dashed line indicates the strength reported pre-exercise. Data are presented as mean±SEM. Significant improvements in DOMS were observed for the PB-H treatment group at 48 h post-exercise following the 12 weeks of supplementation compared with the placebo, whereas only trends were observed in PB-L treatment group.
No differences in muscle soreness were identified in gastrocnemius, gluteus maximus, lower back, or abdominals following supplementation with PB versus placebo. The chronic change in serum antioxidant status following 13 weeks of PB supplementation compared to pre-supplementation values, as measured by FRAP, was significantly different than the change observed in those participants receiving the placebo Fig.
Ferric reducing antioxidant power FRAP following chronic supplementation. Pre-exercise FRAP values are shown pre- and post-supplementation with either a polyphenolic blend PB or a placebo. Creatine kinase CK levels following chronic supplementation. CK was measured pre-exercise, and 15 min and 24, 48, and 96 h post-exercise.
No changes were identified in serum cytokines, 8-isoprostane, LDH, or adrenocorticotropic hormone following chronic supplementation with PB versus the placebo group. Cortisol levels following chronic supplementation. Serum cortisol was assessed pre-exercise, and 15 min and 24, 48, and 96 h post-exercise.
This randomized, double-blind, placebo-controlled clinical study demonstrates that 13 weeks of supplementation with PB improves post-exercise recovery. Supplementation with PB for 13 weeks resulted in significant improvements in serum antioxidant status accompanied by a quicker recovery from a muscle damaging exercise protocol as compared to the placebo treatment.
PB supplementation resulted in a significant attenuation of peak torque losses over 96 h post-exercise, a quicker return of cortisol and CK to pre-exercise values over the 96 h evaluation period, and significantly lower muscle soreness ratings 48 h following the downhill run.
Prior to the current study, no human clinical data existed supporting the ability of tea polyphenols to attenuate post-exercise losses in muscle strength. These findings are supported by a recently published study by Haramizu et al.
These animal data suggest that supplementation with tea polyphenols may diminish post-exercise muscle damage, hasten recovery, and improve physical performance. The animal study conclusions support the findings of the current study which provides the first solid evidence that supplementation with a proprietary blend of tea polyphenols, including catechins and theaflavins, may help athletes overcome post-exercise strength losses brought on by the intense exercise perhaps through the suppressive action of polyphenols on oxidative stress and inflammation.
These findings have critical importance to establishing a scientifically proven and clinically relevant nutritional ingredient that facilitates post-exercise recovery and may have positive implications on athletic performance.
The literature shows that following eccentric exercise reactive oxygen species ROS can activate cell signaling mechanisms resulting in a downstream cascade of events, including elevated serum cortisol and CK 32 , 36 , as observed in the current study.
Data is more limited for cortisol levels post-exercise, but concentrations 1. The reported presence of ROS in muscle tissue post-exercise suggests that antioxidant supplementation may minimize the downstream effects and reduce DOMS after exercise However, despite numerous studies conducted on the use of antioxidant vitamins C and E, either administered in combination or separately, there is no consensus in the literature that these antioxidants are effective in reducing the loss of strength, power, or the onset of DOMS associated with exercise 37 — Polyphenols are a promising group of antioxidants that have been evaluated by researchers 15 , 16 , 22 , 41 — A number of studies indicate the effectiveness of polyphenols, such as catechins, as antioxidants through suppression of NF-κB activation, a key initiator of the cascade of cell signaling, thus showing the potential of catechins to attenuate the activation of exercise-induced inflammation and soreness 44 , In the present study, the downhill run was intended to trigger oxidative stress and the associated post-exercise cascade of events.
This was achieved in all groups as evidenced by losses in strength, increases in both CK and cortisol, and higher muscle soreness ratings immediately post-exercise. CK significantly increased post-exercise peaking at 24 h post-exercise, with no differences between groups, suggesting similar levels of muscle damage and oxidative stress.
However, at 96 h post-exercise, the placebo group had significantly higher CK values, whereas both the PB supplemented groups returned to pre-exercise levels, suggesting a quicker CK clearance and enhanced muscle recovery.
Similar findings were also obtained for cortisol, corroborating the effects seen with CK following chronic PB supplementation. Cortisol responses are directly related to exercise intensity and duration, increasing post-exercise Although no between group differences were identified at earlier time points, circulating cortisol in the placebo group remained significantly higher at 96 h compared to the PB-H group.
Studies have shown that supplementation with either N-acetyl cysteine or EGCG alone in active men did not affect cortisol levels over a 72 h post-exercise recovery period However, supplementation with theaflavin-enriched black tea extract has been shown to lower overall cortisol secretion within the first h post-exercise In the current study, the subsequent continued exercise on days 1, 2, and 4 may have allowed differences in cortisol and oxidative stress responses to be identified.
Due to of the catabolic nature of cortisol, the ability of PB to significantly reduce cortisol secretion post-exercise may facilitate improved recovery and decreased muscle stress. Although initial elevations in cortisol may be beneficial, prolonged elevations in cortisol can lead to chronic inflammation.
Excessive cortisol secretion in response to exercise may contribute to overtraining 3. One of the major deterrents to performing subsequent activities after an intense bout of exercise is DOMS.
Although an eccentric exercise was used as a trigger to inflict muscle soreness in the current study, DOMS is often felt after vigorous exercise.
Compared to placebo, PB-H supplementation significantly blunted the increase in both whole body and hamstring muscle soreness ratings at 48 h post-exercise. The individuals in the current study were not sedentary, reporting amongst all groups approximately 5 h of activity per week at screening.
The more active subjects used in the current study may have displayed enhanced tolerance to DOMS and blunted physiological responses after training, thereby impairing our ability to detect greater changes in DOMS and markers of muscle damage or catabolism After an intense or unaccustomed exercise bout, secondary damage associated with inflammatory processes may occur over several days following the initial insult Research has focused on nutritional strategies aimed at reducing the oxidative stress and inflammation traditionally associated with fatigue and impaired recovery post-exercise.
Although changes in CK, cortisol, and muscle soreness were observed after the downhill run, several markers of inflammation, IL-6, IL, TNF-α, and isoprostanes did not significantly increase after the downhill run.
However, this difficulty or inability to detect changes at the circulating level suggests future work should incorporate muscle biopsies. An additional confounding factor is that trained subjects often display attenuated stress response to exercise challenges, which has been attributed to several physiological adaptations including increased heat shock protein levels or an enhanced antioxidant capacity In addition, the ability to accurately assess changes may have been missed due to the time points chosen for analyses, which were not ideal for all biomarkers 15 min, and 24, 48, and 96 h post-exercise.
Grape extract was able to limit the reduction of FRAP in elite athletes during training compared to the placebo group Green tea extract significantly increased plasma FRAP in the context of resistance training workouts after 14 days and showed a trend after 7 days 50 , Although the active constituents of PB were not directly measured in blood in the current study, the significant chronic increases in serum FRAP in the treatment groups over placebo support that the supplement was bioavailable.
The ability to increase serum antioxidant status may minimize the oxidative stress-induced cell signaling post-exercise, thus positively affecting biomarkers such as CK and cortisol and ultimately muscle soreness and performance during the recovery period.
The performance strength testing used is regarded as the best functional marker of muscle damage aside from muscle biopsy collection which is both invasive and expensive Furthermore, participants were introduced to the protocol through three familiarization sessions during the screening period to minimize any potential learning effects.
Still another asset of the current study is that strength recovery, blood biomarkers, and muscle soreness were all measured simultaneously. Arent et al.
Kelli A. Chirouzes Probiotics for womens health Michael A. Background : Fat burning exercises can initiate a cascade of MMuscle and oxidative Msucle events leading to delayed onset muscle soreness. Polyphenols possess antioxidant and anti-inflammatory properties. Objective : The current study examined the effects of a proprietary polyphenolic blend PBcontaining catechins and theaflavins, on exercise performance and recovery following an eccentric exercise challenge. Jasser El Catechinz, Min-Ho Oak, Fat burning exercises Anglard, Muscle recovery catechins B. Objective : Regular consumption of green tea is recovvery with Mucsle reduced Matcha green tea weight loss Muscle recovery catechins mortality due to coronary diseases recovety cancer. Musclee present study examined whether a catechkns tea extract GTE Muscle recovery catechins activation of catechinz metalloproteinase-2 MMP-2a major collagenase involved in vascular remodeling of atherosclerotic plaques, in vascular smooth muscle cells VSMCs. Methods and results : The expression of MMP-2 was assessed by Northern and Western blot analyses in human aortic VSMCs. MMP-2 activity was evaluated by zymography, membrane-type1-MMP MT1-MMP, MMP activity by an enzymatic assay, and cell invasion by a modified Boyden chamber assay. GTE reduced the expression of MMP-2 mRNA and protein. GTE, EGCG and ECG directly inhibited cell-associated MT1-MMP activity, the physiological activator of MMP-2, in a reversible manner.
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