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Body fluids and electrolytesElecfrolyte physiology provides an integral description for mechanisms of Electrolyte Homeostasis entire spectrum of Homeostqsis in the Electroyte body. Successful work of the mechanisms requires a highly Homeostaeis internal environment, which is continuously Electrolyte Homeostasis by Honey glazed sweet potatoes organs under the influence of nerve impulses, hormones, Hmeostasis, and autacoids.
The Epectrolyte discusses the physicochemical parameters of the human internal environment and Electrolytf possibilities of clearance methods Natural ways to prevent cancer the involvement of kidneys in homeostatic processes.
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Google Scholar. Natochin, Yu. Nauk, Injury prevention in swimming. Chernigovskaya, T. Homeosgasis, Electrolyte Homeostasis. Petersburg: Inst.
Mozga Electroyte. Nauk, Bernard, C. Ballière, Book Electrrolyte Scholar. Electrlyte, W. Article Google Ekectrolyte.
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Electrklyte, V. Natural ways to prevent cancer i Electroyte signalizatsiya: Homeostzsis posobie OHmeostasis of Molecular Endocrinology. Review and Intracellular Signaling: ManualMoscow: GEOTAR-Media, Nadkarni, P.
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Golosova, D. and Natochin, Yu. Samoilov, A. Pavlov: Selected Articles and SpeechesMoscow: Akad. Nauk SSSR, and Chernigovskaya, T. Pavlov, I. Trophic Innervation: Selected Research WorksNatochin, Yu.
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You can also search for this author in PubMed Google Scholar. Correspondence to Yu. Original Russian Text © Yu. Natochin,published in Fiziologiya Cheloveka,Vol. Reprints and permissions. Human Physiology: Water and Electrolyte Homeostasis. Hum Physiol 44— Download citation. Received : 19 Homeeostasis Published : 27 June Issue Date : May Anyone you share the following link with will be able to read this Electroyte.
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Abstract Human physiology provides an integral description for mechanisms of the entire spectrum of functions in the human body.
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Google Homeostwsis Bernard, C. Book Google Scholar Cannon, W. Article Google Scholar Barcroft, J. PubMed CAS Google Scholar Noble, D.
Article PubMed Google Scholar Guyton, A. Google Scholar Grigoriev, A. Article PubMed CAS Google Scholar Tkachuk, V. Google Scholar Nadkarni, P. Article PubMed PubMed Central CAS Google Scholar Marina, A. Article PubMed CAS Google Scholar Natochin, Y.
Article PubMed CAS Google Scholar Hao, C. Article PubMed CAS Google Scholar Natochin, Yu. Google Scholar Golosova, D. Article PubMed CAS Google Scholar Samoilov, A. Article Google Scholar Pavlov, I. Google Scholar Natochin, Y. Article PubMed Google Scholar Download references. Author information Authors and Affiliations Sechenov Institute Electroylte Evolutionary Physiology and Biochemistry, Russian Academy of Sciences, St.
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: Electrolyte Homeostasis| We have a new app! | Please Wait. Spreca A, Simonetti S, Rambotti MG. The thirst Natural ways to prevent cancer begins when osmoreceptors Homeostasix a decrease in water levels in Natural ways to prevent cancer Electrolytw. Quantitative data are presented. Añadir al carrito. The serine protease Corin is a novel modifier of the Agouti pathway. Compared to WT mice, however, corin KO mice had reduced sweat excretion, as indicated by smaller black-staining areas in footpads, analyzed at 2 min after pilocarpine injection 3. |
| Essential Fluid, Electrolyte and Ph Homeostasis | Caverzasio J, Bonjour JP, Fleisch H Adaptation of tubular phosphate transport: relation between phosphate requirement as influenced by growth and supply. Dehydration There are three types of dehydration: Hypotonic or hyponatremic primarily a loss of electrolytes, sodium in particular. Karlberg BE, Ottosson AM Acromegaly and hypertension: role of the renin-angiotensin system. Home Electrolyte Homeostasis. Gene Ontology analysis of cluster 8 determined this cluster is significantly enriched with genes associated with the biological pathways: cell cycle, chromosomal segregation, mitotic nuclear division, cell division, and microtubule-based movement Figure 6B. Pediatr Res — A limitation to our transcriptome analysis is that the direct targets of HDAC1 and HDAC2 cannot be distinguished from indirect targets. |
| 25.3A: Sodium, Electrolytes, and Fluid Balance | Hojeostasis Condiciones de venta Quiénes Somos Aviso Contacto. This Metabolic support for nutrient absorption an open access article distributed under the Natural ways to prevent cancer of the Creative Commons Electrolyte Homeostasis LicenseNatural ways to prevent cancer permits Homeostaiss use, Homeostasos, and reproduction in any medium, provided the original author and source are credited. Primers used in the study are listed in Table A. Representative photos and quantitative data are shown. A normal IgG was used as a negative control in immunohistochemical analysis. Biglieri EG, Watlington CO, Forsham PH Sodium retention with human growth hormone and its subfractions. gov: Water-Electrolyte Imbalance National Institutes of Health. |
| Renal effects of growth hormone. II. Electrolyte homeostasis and body composition | Username Error: Please enter Eldctrolyte Name. Eleftrolyte renal Natural ways to prevent cancer Anti-hypertensive nutritional supplements contributes to sodium retention in proteinuric kidney diseases. However, HSW rats with MS infusion had a significant Electrllyte in urinary sodium excretion Homfostasis Natural ways to prevent cancer 7 compared with vehicle-treated HSW Homeostasks Supplemental Electrolyte Homeostasis Electtrolyteyet plasma sodium appeared normal in this group of rats Supplemental Table 4. J Clin Endocrinol Metab — Google Scholar Burstein S, Chen I-W, Tsang RC Effects of growth hormone replacement therapy on 1,dihydroxyvitamin D and calcium metabolism. J Endocrinol — Google Scholar Simone PG, Solomon S Aldosterone and growth hormone: influence of diet and hypophysectomy on rat renal response. Sweat gland progenitors in development, homeostasis, and wound repair. C Co-staining of corin green and SMA red in the secretory epithelial cells. |
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Jasin HE, Fink CW, Wise W, Ziff M Relationship between urinary hydroxyproline and growth. J Clin Nutr — Johansen JS, Pederson SA, Jorgensen JO, Riis BJ, Christiansen C, Christiansen JS, Skakkebaek NE Effects of growth hormone GH on plasma bone G1a protein in GH-deficient adults.
Markowitz ME, Dimartino-Nardi J, Gasparini F, Fishman K, Rosen JF, Saenger P Effects of growth hormone therapy on circadian osteocalcin rhythms in idiopathic short stature.
Shore RM, Chesney RW, Mazess RB, Rose PG, Bargman GJ Bone mineral status in growth hormone deficiency. J Pediatr — Greig F, AvRuskin TW, Greenfield E, Prasad V, Bastian W, Castello S Change in bone density in children during treatment with growth hormone abstract.
Horm Res 31 [Suppl 1] Kaufman JM, Vandeweghe M, Taelman P, Craen R Bone mineral status in GH-deficient male patients with isolated and multiple deficiencies abstract. Halse J, Melsen F, Mosekilde L Iliac crest bone mass and remodelling in acromegaly.
Diamond T, Nery L, Posen S Spinal and peripheral bone mineral densities in acromegaly: the effects of excess growth hormone and hypogonadism. Ann Intern Med — Halse J, Gordeladza JO Urinary hydroxyproline excretion in acromegaly. De La Piedra C, Larranaga ECJ, Castro N, Horcajada C, Rapado A, Herrera Pombo JL Correlation among plasma osteocalcin, growth hormone, and somatomedin C in acromegaly.
Kainer G, Nakano M, Massie FS, Foreman JW, Chan JCM Severe hypercalciuria: an unexpected result of concurrent use of 1,dihydroxy-vitamin D3 and growth hormone in uremic rats. Pediatr Res — Parfitt AM Bone as a source of urinary calcium — osseus hypercalciuria. In: Coe FL ed Hypercalciuric states.
Pathogenesis, consequences and treatment. Grune Stratton, Orlando, pp — Yamamoto T A case of active acromegaly with reduced height and type 1 renal tubular acidosis. Endocrinol Jpn — Tonshoff B, Heinrich U, Mehls O How safe is the treatment of uraemic children with recombinant human growth hormone.
Pediatr Nephrol — Rudman D, Kutner MM, Blackston RD, Jansen RD, Patterson JH Normal variant short stature: subclassification based on responses to exogenous human growth hormone.
Dahms WT, Owens RP, Kalhnan SC, Kerr DS, Danish RK Urea synthesis, nitrogen balance and glucose turnover in growth-hormone-deficient children before and after growth hormone administration.
Daughaday WH Growth hormone: normal synthesis, secretion, control, and mechanisms of action. In: De Groot LJ ed Endocrinology. Saunders, Philadelphia, pp — McLellan AR, Connell JMC, Beastall GH, Teasdale G, Davies DL Growth hormone, body composition and somatomedin C after treatment of acromegaly.
Q J Med — Bengtsson B-A, Rosen T Body composition in patients treated for hypopituitarism. Acta Paediatr Scand [Suppl] Tanner JM, Hughes PCR, Whitehouse RH Comparative rapidity of response of height, limb muscle and limb fat to treatment with human growth hormone in patients with and without growth hormone deficiency.
Parra A, Argote RM, Garcia G, Cervantes C, Alatorre SA, Perez-Pasten E Body composition in hypopituitary dwarfs before and during human growth hormone therapy.
Zachmann M, Fernandez F, Tassinari D, Thakker R, Prader A Anthropometric measurements in patients with growth hormone deficiency before treatment with human growth hormone. Eur J Pediatr — Rosenbaum M, Gerner JM, Leibel RL Effects of systemic growth hormone GH administration on regional adipose tissue distribution and metabolism in GH-deficient children.
Ballard FJ, Burgoyne JL, Tomas FM, Penfold JL Growthhormone induced changes in myofibrillar protein breakdown in hypopituitary children. Clin Sci — Lee PA, Blizzard RM, Cheek DB, Holt AB Growth and body composition in intrauterine growth retardation IUGR before and during human growth hormone administration.
Walker JM, Bond SA, Voss LD, Betts PR, Wootton SA, Jackson AA Treatment of short normal children with growth hormone — a cautionary tale? Lancet — Koch VH, Lippe BM, Nelson PA, Boechat MI, Sherman BM, Fine RN Accelerated growth after recombinant human growth hormone treatment of children with chronic renal failure.
Crist DM, Peake GT, Egan PA, Waters DL Body composition response to exogenous GH during training in highly conditioned adults. J Appl Physiol — Lippe B, Frasier DS How should we test for growth hormone deficiency, and whom should we treat?
Johansson G, Sietnieks A, Janssens F, Proesmans W, Vanderschueren-Lodeweyckx M, Holmberg C, Sipila I, Broyer M, Rappaport R, Albertsson-Wikland K, Berg U, Jodal U, Rees L, Rigden SPA, Preece MA Recombinant human growth hormone treatment in short children with chronic renal disease, before transplantation or with functioning renal transplants: an interim report on five European studies.
Acta Paediatr Scand [Suppl] — Download references. Department of Nephrology, Prince of Wales Children's Hospital, High Street, Randwick, , Sydney, New South Wales, Australia. Graham D. Ogle, Andrew R. You can also search for this author in PubMed Google Scholar.
Reprints and permissions. Ogle, G. Renal effects of growth hormone. Electrolyte homeostasis and body composition. Pediatr Nephrol 6 , — Download citation.
Received : 03 December Revised : 15 January Accepted : 16 January Issue Date : September Anyone you share the following link with will be able to read this content:.
Sorry, a shareable link is not currently available for this article. Provided by the Springer Nature SharedIt content-sharing initiative. Abstract Growth hormone GH , either directly or through insulin-like growth factor-1 IGF-1 , has a wide spectrum of physiological and renal effects.
Access this article Log in via an institution. References Henneman PH, Forbes AP, Moldawer M, Dempsey EF, Carroll EL Effects of human growth hormone in man. J Clin Invest — Google Scholar Glafkides CM, Bennett LL Reduction of urinary sodium and potassium of diabetic rats produced by hypophyseal growth hormone.
Proc Soc Exp Biol Med — Google Scholar Stein JD, Bennett LL, Batts AA, Hao LIC Sodium, potassium and chloride retention produced by growth hormone in the absence of the adrenals. Am J Physiol — Google Scholar Biglieri EG, Watlington CO, Forsham PH Sodium retention with human growth hormone and its subfractions.
J Clin Endocrinol Metab — Google Scholar Manson J MK, Wilmore DW Positive nitrogen balance with human growth hormone and hypocaloric intravenous feeding. Surgery — Google Scholar Beck JC, McGarry EE, Dyrenfurth I, Morgen O, Bird ED, Venning EH Primate growth hormone studies in man.
Metab Clin Exp — Google Scholar Binnerts A, Wilson JHP, Lamberts SWJ The effects of human growth hormone administration in elderly adults with recent weight loss.
J Clin Endocrinol Metab — Google Scholar Ho KY, Weissberger AJ, Stuart MC, Day RO, Lazarus L The pharmacokinetics, safety and endocrine effects of authentic biosynthetic human growth hormone in normal subjects. Clin Endocrinol Oxf — Google Scholar Ho K, Weissberger AJ The antinatriuretic action of biosynthetic human growth hormone in man involves activation of the renin-angiotensin system.
Metabolism — Google Scholar Humatrope Prescribing Information Eli-Lilly, Australia Salomon F, Cuneo RC, Hesp R, Soensken PM The effects of treatment with recombinant human growth hormone on body composition and metabolism in adults with growth hormone deficiency.
N Engl J Med — Google Scholar Clemmons DR, Snyder DK, Williams R, Underwood LE Growth hormone administration conserves lean body mass during dietary restriction in obese subjects. J Clin Endocrinol Metab — Google Scholar Rudman D, Feller AG, Nagaraj MS, Gergans GA, Lalitha PY, Goldberg AF, Schlenker RA, Cohn L, Rudman IW, Mattson DE Effects of human growth hormone in men over 60 years old.
N Engl J Med —6 Google Scholar Ludens JM, Bach RR, Williamson HE Characteristics of the antinatriuretic action of growth hormone. Proc Soc Exp Biol Med — Google Scholar Venning EH, Dyrenfurth I, Giroud CIP, Beck JC Effect of growth hormone on aldosterone excretion.
Metabolism — Google Scholar Penhoat A, Chatelain PG, Jaillard C, Saez JM Characterization of insulin-like factor-1 and insulin receptors on cultured bovine adrenal fasciculata cells. Electrolytes in living systems include sodium, potassium, chloride, bicarbonate, calcium, phosphate, magnesium, copper, zinc, iron, manganese, molybdenum, copper, and chromium.
In terms of body functioning, six electrolytes are most important: sodium, potassium, chloride, bicarbonate, calcium, and phosphate. These six ions aid in nerve excitability, endocrine secretion, membrane permeability, buffering body fluids, and controlling the movement of fluids between compartments.
Excretion of ions occurs mainly through the kidneys, with lesser amounts lost in sweat and in feces. Excessive sweating may cause a significant loss, especially of sodium and chloride. Severe vomiting or diarrhea will cause a loss of chloride and bicarbonate ions.
Adjustments in respiratory and renal functions allow the body to regulate the levels of these ions in the ECF. The following table lists the reference values for blood plasma, cerebrospinal fluid CSF , and urine for the six ions addressed in this section.
In a clinical setting, sodium, potassium, and chloride are typically analyzed in a routine urine sample. In contrast, calcium and phosphate analysis requires a collection of urine across a hour period, because the output of these ions can vary considerably over the course of a day. Urine values reflect the rates of excretion of these ions.
Imbalances of these ions can result in various problems in the body, and their concentrations are tightly regulated.
Aldosterone and angiotensin II control the exchange of sodium and potassium between the renal filtrate and the renal collecting tubule.
Calcium and phosphate are regulated by PTH, calcitrol, and calcitonin. Sodium is the major cation of the extracellular fluid. It is responsible for one-half of the osmotic pressure gradient that exists between the interior of cells and their surrounding environment.
This excess sodium appears to be a major factor in hypertension high blood pressure in some people. Excretion of sodium is accomplished primarily by the kidneys. Sodium is freely filtered through the glomerular capillaries of the kidneys, and although much of the filtered sodium is reabsorbed in the proximal convoluted tubule, some remains in the filtrate and urine, and is normally excreted.
Hyponatremia is a lower-than-normal concentration of sodium, usually associated with excess water accumulation in the body, which dilutes the sodium.
An abnormal loss of sodium from the body can result from several conditions, including excessive sweating, vomiting, or diarrhea; the use of diuretics; excessive production of urine, which can occur in diabetes; and acidosis, either metabolic acidosis or diabetic ketoacidosis.
At the cellular level, hyponatremia results in increased entry of water into cells by osmosis, because the concentration of solutes within the cell exceeds the concentration of solutes in the now-diluted ECF. The excess water causes swelling of the cells; the swelling of red blood cells—decreasing their oxygen-carrying efficiency and making them potentially too large to fit through capillaries—along with the swelling of neurons in the brain can result in brain damage or even death.
Hypernatremia is an abnormal increase of blood sodium. It can result from water loss from the blood. Hormonal imbalances involving ADH and aldosterone may also result in higher-than-normal sodium values. Potassium is the major intracellular cation.
It helps establish the resting membrane potential in neurons and muscle fibers after membrane depolarization and action potentials.
In contrast to sodium, potassium has very little effect on osmotic pressure. Hypokalemia is an abnormally low potassium blood level. Similar to the situation with hyponatremia, hypokalemia can occur because of either an absolute reduction of potassium in the body or a relative reduction of potassium in the blood due to the redistribution of potassium.
An absolute loss of potassium can arise from decreased intake, frequently related to starvation. It can also come about from vomiting, diarrhea, or alkalosis. Hyperkalemia , an elevated potassium blood level, also can impair the function of skeletal muscles, the nervous system, and the heart.
Hyperkalemia can result from increased dietary intake of potassium. In such a situation, potassium from the blood ends up in the ECF in abnormally high concentrations.
This can result in a partial depolarization excitation of the plasma membrane of skeletal muscle fibers, neurons, and cardiac cells of the heart, and can also lead to an inability of cells to repolarize.
Because of such effects on the nervous system, a person with hyperkalemia may also exhibit mental confusion, numbness, and weakened respiratory muscles.
Chloride is the predominant extracellular anion. Chloride is a major contributor to the osmotic pressure gradient between the ICF and ECF, and plays an important role in maintaining proper hydration. Hypochloremia , or lower-than-normal blood chloride levels, can occur because of defective renal tubular absorption.
Vomiting, diarrhea, and metabolic acidosis can also lead to hypochloremia. Hyperchloremia , or higher-than-normal blood chloride levels, can occur due to dehydration, excessive intake of dietary salt NaCl or swallowing of sea water, aspirin intoxication, congestive heart failure, and the hereditary, chronic lung disease, cystic fibrosis.
In people who have cystic fibrosis, chloride levels in sweat are two to five times those of normal levels, and analysis of sweat is often used in the diagnosis of the disease. Bicarbonate is the second most abundant anion in the blood. About two pounds of calcium in your body are bound up in bone, which provides hardness to the bone and serves as a mineral reserve for calcium and its salts for the rest of the tissues.
Teeth also have a high concentration of calcium within them. A little more than one-half of blood calcium is bound to proteins, leaving the rest in its ionized form.
In addition, calcium helps to stabilize cell membranes and is essential for the release of neurotransmitters from neurons and of hormones from endocrine glands. Calcium is absorbed through the intestines under the influence of activated vitamin D.
A deficiency of vitamin D leads to a decrease in absorbed calcium and, eventually, a depletion of calcium stores from the skeletal system, potentially leading to rickets in children and osteomalacia in adults, contributing to osteoporosis. Hypocalcemia , or abnormally low calcium blood levels, is seen in hypoparathyroidism, which may follow the removal of the thyroid gland, because the four nodules of the parathyroid gland are embedded in it.
Hypercalcemia , or abnormally high calcium blood levels, is seen in primary hyperparathyroidism. Some malignancies may also result in hypercalcemia. Phosphate is found in phospholipids, such as those that make up the cell membrane, and in ATP, nucleotides, and buffers. Hypophosphatemia , or abnormally low phosphate blood levels, occurs with heavy use of antacids, during alcohol withdrawal, and during malnourishment.
In the face of phosphate depletion, the kidneys usually conserve phosphate, but during starvation, this conservation is impaired greatly. Hyperphosphatemia , or abnormally increased levels of phosphates in the blood, occurs if there is decreased renal function or in cases of acute lymphocytic leukemia.
Additionally, because phosphate is a major constituent of the ICF, any significant destruction of cells can result in dumping of phosphate into the ECF. Official websites use. gov A.
gov website belongs to an official government organization in the United States. gov website. Share sensitive information only on official, secure websites. Electrolytes are minerals that have an electric charge when they are dissolved in water or body fluids, including blood.
The electric charge can be positive or negative. You have electrolytes in your blood, urine pee , tissues, and other body fluids. An electrolyte imbalance means that the level of one or more electrolytes in your body is too low or too high.
It can happen when the amount of water in your body changes. The amount of water that you take in should equal the amount you lose. If something upsets this balance, you may have too little water dehydration or too much water overhydration. Some of the more common reasons why you might have an imbalance of the water in your body include:.
A test called an electrolyte panel can check the levels of your body's main electrolytes. A related test, the anion gap blood test , checks whether your electrolytes are out of balance or if your blood is too acidic or not acidic enough. The treatment for an electrolyte imbalance depends on which electrolytes are out of balance, if there is too little or too many, and what is causing the imbalance.
In minor cases, you may just need to make some changes to your diet. In other cases, you may need other treatments. For example:. The information on this site should not be used as a substitute for professional medical care or advice.
Contact a health care provider if you have questions about your health. Fluid and Electrolyte Balance.
If your institution Skincare for mature skin to this Electtolyte, and you don't have an Access Natural ways to prevent cancer, please contact your library's reference Natural ways to prevent cancer for information Homoestasis how to gain access Homeostaasis this resource from Electrolyte Homeostasis. Take the Access library with you wherever you go—easy access to books, videos, images, podcasts, personalized features, and more. Download the Access App here: iOS and Android. Learn more here! Please consult the latest official manual style if you have any questions regarding the format accuracy. Disorders of sodium and water, calcium, phosphorus, potassium, and magnesium homeostasis are addressed separately in this chapter.Electrolyte Homeostasis -
In: De Groot LJ ed Endocrinology. Saunders, Philadelphia, pp — McLellan AR, Connell JMC, Beastall GH, Teasdale G, Davies DL Growth hormone, body composition and somatomedin C after treatment of acromegaly.
Q J Med — Bengtsson B-A, Rosen T Body composition in patients treated for hypopituitarism. Acta Paediatr Scand [Suppl] Tanner JM, Hughes PCR, Whitehouse RH Comparative rapidity of response of height, limb muscle and limb fat to treatment with human growth hormone in patients with and without growth hormone deficiency.
Parra A, Argote RM, Garcia G, Cervantes C, Alatorre SA, Perez-Pasten E Body composition in hypopituitary dwarfs before and during human growth hormone therapy. Zachmann M, Fernandez F, Tassinari D, Thakker R, Prader A Anthropometric measurements in patients with growth hormone deficiency before treatment with human growth hormone.
Eur J Pediatr — Rosenbaum M, Gerner JM, Leibel RL Effects of systemic growth hormone GH administration on regional adipose tissue distribution and metabolism in GH-deficient children. Ballard FJ, Burgoyne JL, Tomas FM, Penfold JL Growthhormone induced changes in myofibrillar protein breakdown in hypopituitary children.
Clin Sci — Lee PA, Blizzard RM, Cheek DB, Holt AB Growth and body composition in intrauterine growth retardation IUGR before and during human growth hormone administration. Walker JM, Bond SA, Voss LD, Betts PR, Wootton SA, Jackson AA Treatment of short normal children with growth hormone — a cautionary tale?
Lancet — Koch VH, Lippe BM, Nelson PA, Boechat MI, Sherman BM, Fine RN Accelerated growth after recombinant human growth hormone treatment of children with chronic renal failure. Crist DM, Peake GT, Egan PA, Waters DL Body composition response to exogenous GH during training in highly conditioned adults.
J Appl Physiol — Lippe B, Frasier DS How should we test for growth hormone deficiency, and whom should we treat? Johansson G, Sietnieks A, Janssens F, Proesmans W, Vanderschueren-Lodeweyckx M, Holmberg C, Sipila I, Broyer M, Rappaport R, Albertsson-Wikland K, Berg U, Jodal U, Rees L, Rigden SPA, Preece MA Recombinant human growth hormone treatment in short children with chronic renal disease, before transplantation or with functioning renal transplants: an interim report on five European studies.
Acta Paediatr Scand [Suppl] — Download references. Department of Nephrology, Prince of Wales Children's Hospital, High Street, Randwick, , Sydney, New South Wales, Australia.
Graham D. Ogle, Andrew R. You can also search for this author in PubMed Google Scholar. Reprints and permissions. Ogle, G. Renal effects of growth hormone. Electrolyte homeostasis and body composition.
Pediatr Nephrol 6 , — Download citation. Received : 03 December Revised : 15 January Accepted : 16 January Issue Date : September Anyone you share the following link with will be able to read this content:.
Sorry, a shareable link is not currently available for this article. Provided by the Springer Nature SharedIt content-sharing initiative.
Abstract Growth hormone GH , either directly or through insulin-like growth factor-1 IGF-1 , has a wide spectrum of physiological and renal effects.
Access this article Log in via an institution. References Henneman PH, Forbes AP, Moldawer M, Dempsey EF, Carroll EL Effects of human growth hormone in man. J Clin Invest — Google Scholar Glafkides CM, Bennett LL Reduction of urinary sodium and potassium of diabetic rats produced by hypophyseal growth hormone.
Proc Soc Exp Biol Med — Google Scholar Stein JD, Bennett LL, Batts AA, Hao LIC Sodium, potassium and chloride retention produced by growth hormone in the absence of the adrenals. Am J Physiol — Google Scholar Biglieri EG, Watlington CO, Forsham PH Sodium retention with human growth hormone and its subfractions.
J Clin Endocrinol Metab — Google Scholar Manson J MK, Wilmore DW Positive nitrogen balance with human growth hormone and hypocaloric intravenous feeding. Surgery — Google Scholar Beck JC, McGarry EE, Dyrenfurth I, Morgen O, Bird ED, Venning EH Primate growth hormone studies in man.
Metab Clin Exp — Google Scholar Binnerts A, Wilson JHP, Lamberts SWJ The effects of human growth hormone administration in elderly adults with recent weight loss.
J Clin Endocrinol Metab — Google Scholar Ho KY, Weissberger AJ, Stuart MC, Day RO, Lazarus L The pharmacokinetics, safety and endocrine effects of authentic biosynthetic human growth hormone in normal subjects.
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N Engl J Med — Google Scholar Clemmons DR, Snyder DK, Williams R, Underwood LE Growth hormone administration conserves lean body mass during dietary restriction in obese subjects. J Clin Endocrinol Metab — Google Scholar Rudman D, Feller AG, Nagaraj MS, Gergans GA, Lalitha PY, Goldberg AF, Schlenker RA, Cohn L, Rudman IW, Mattson DE Effects of human growth hormone in men over 60 years old.
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